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Pekas -

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Korean Journal Sports Medicine





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It is well-accepted that vascular dysfunction plays a key role in the pathophysiology of cardiovascular diseases. Although vascular dysfunction is multi-faceted, exercise is a commonly recommended prophylactic strategy to preserve vascular function. We and others have shown that exercise training can elicit beneficial effects on vascular function (e.g., blood pressure and conduit artery function) in healthy and clinical populations1-4. In fact, indices of vascular function are enhanced shortly after acute exercise5,6, suggesting that the postexercise recovery period may be a crucial component for facilitating long-term vascular adaptations7. Poor habits may be detrimental to this recovery window, such as cigarette smoking. Cigarette smoking is considered a common modifiable risk factor for cardiovascular diseases and is associated with arterial stiffness and endothelial dysfunction8,9. Since exercise is often recommended to individuals with cardiovascular risk factors to prevent disease, it is imperative to understand how smoking can impact acute exercise recovery. Previous studies have investigated cigarette smoking prior to acute exercise and revealed that this can impair normal vascular and exercise pressor responses, thus inducing greater cardiac and arterial strain10-12. However, until recently, the impacts of cigarette smoking on hemodynamics and conduit artery function during recovery after aerobic exercise have not been explored. In this issue of The Korean Journal of Sports Medicine, Cho et al.13 investigated the effects of cigarette smoking on blood pressure and conduit artery function during recovery after an acute bout of moderate-intensity aerobic exercise.


This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License


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Creative Commons Attribution-Noncommercial 4.0 License
This work is licensed under a Creative Commons Attribution-Noncommercial 4.0 License