![2017 Student Research and Creative Activity Fair](../../../../assets/md5images/f4989b42bf549b445c1752c7648a8243.png)
Advisor Information
Dustin Slivka
Location
Dr. C.C. and Mabel L. Criss Library
Presentation Type
Poster
Start Date
3-3-2017 12:30 PM
End Date
3-3-2017 1:45 PM
Abstract
There has been recent debate on the difference in physiological response between exposure to normobaric hypoxia (oxygen fraction altered) and hypobaric hypoxia (barometric pressure altered). Purpose: To determine the effects of normobaric normoxia (NN), hypobaric hypoxia (HH), and normobaric hypoxia (NH) on heart rate and arterial oxygen saturation (SaO2) after exercise. Methods: Fifteen recreationally active participants (eight males and seven females) completed three separate 1 h cycling bouts at 70% of their peak aerobic capacity. Following each trial, participants recovered in the supine position for 4 h in NN (975 m), HH (4420 m), or NH (4420 m). SaO2 was collected pre-, mid-, and post- exercise, as well as in each hour of subsequent recovery. Heart rate was monitored continuously throughout the trials. Results: There were no differences in SaO2 or heart rate pre-, mid- or immediately post-exercise between the three trials (p > 0.05). However, during recovery, SaO2 was lower in the HH and NH compared to NN (p < 0.001), and HH was lower than NH in hours 1, 3 and 4. During recovery, heart rate remained elevated in both hypoxic conditions relative to NN (p < 0.05), and heart rate in HH remained elevated relative to NH. Conclusion: These data indicate that post-exercise heart rate and SaO2 are influenced by the method of achieving hypoxia and that caution should be made when translating results between these methods. Further research is needed to examine how these recovery parameters may influence performance and other physiological parameters.
Effects of Hypobaric and Normobaric Hypoxia on Heart Rate and Blood Oxygen Saturation
Dr. C.C. and Mabel L. Criss Library
There has been recent debate on the difference in physiological response between exposure to normobaric hypoxia (oxygen fraction altered) and hypobaric hypoxia (barometric pressure altered). Purpose: To determine the effects of normobaric normoxia (NN), hypobaric hypoxia (HH), and normobaric hypoxia (NH) on heart rate and arterial oxygen saturation (SaO2) after exercise. Methods: Fifteen recreationally active participants (eight males and seven females) completed three separate 1 h cycling bouts at 70% of their peak aerobic capacity. Following each trial, participants recovered in the supine position for 4 h in NN (975 m), HH (4420 m), or NH (4420 m). SaO2 was collected pre-, mid-, and post- exercise, as well as in each hour of subsequent recovery. Heart rate was monitored continuously throughout the trials. Results: There were no differences in SaO2 or heart rate pre-, mid- or immediately post-exercise between the three trials (p > 0.05). However, during recovery, SaO2 was lower in the HH and NH compared to NN (p < 0.001), and HH was lower than NH in hours 1, 3 and 4. During recovery, heart rate remained elevated in both hypoxic conditions relative to NN (p < 0.05), and heart rate in HH remained elevated relative to NH. Conclusion: These data indicate that post-exercise heart rate and SaO2 are influenced by the method of achieving hypoxia and that caution should be made when translating results between these methods. Further research is needed to examine how these recovery parameters may influence performance and other physiological parameters.