Spanning paradigms: Using immunotherapy to enhance human natural killer cell antibody dependent cell-mediated cytotoxicity against cancer and infectious disease

Presenter Type

UNO Undergraduate Student

Major/Field of Study

Biology

Other

Molecular & Biomedical Biology/Medical Humanities

Author ORCID Identifier

0009-0007-0587-5948

Advisor Information

Paul W. Denton PhD.

Location

CEC RM #231

Presentation Type

Oral Presentation

Poster Size

N/A

Start Date

22-3-2024 10:30 AM

End Date

22-3-2024 11:45 AM

Abstract

A primary goal of the Denton Immunobiology laboratory is to evaluate immunotherapy strategies with the goal of improving the killing capacity of human natural killer (NK) cells. To date, this work has been in the context of killing cancer cells. Malignancy is a disease paradigm with a nearly immeasurable scope. However, another disease paradigm has a similarly large sphere – infectious diseases. This project's goal is to determine whether immunotherapy findings in a cancer context can span paradigms and similarly impact treatment approaches in an infectious disease context. Our goal is to perform infectious disease-related experiments without the need to incorporate fully infectious agents into our experimental approach. To do this, we obtained cells that express human immunodeficiency virus (HIV) envelope proteins constitutively. HIV was chosen as the pathogen to represent the infectious disease paradigm in our studies because HIV treatments exist but are not curative (yet). Our goal is to contribute to many research efforts focused on helping to “train” the immune system to fight HIV in the absence of other treatments (e.g., antiretroviral therapy). The target cells with HIV protein on their surface appear to be infected from the perspective of the human NK cells that we have treated with immunotherapies. To allow the NK cells to recognize the presence of HIV protein, we use an antibody that is specific for the envelope and capable of directing the NK cell to perform the killing function known as antibody-dependent cell-mediated cytotoxicity (ADCC). Data to date will be presented.

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Mar 22nd, 10:30 AM Mar 22nd, 11:45 AM

Spanning paradigms: Using immunotherapy to enhance human natural killer cell antibody dependent cell-mediated cytotoxicity against cancer and infectious disease

CEC RM #231

A primary goal of the Denton Immunobiology laboratory is to evaluate immunotherapy strategies with the goal of improving the killing capacity of human natural killer (NK) cells. To date, this work has been in the context of killing cancer cells. Malignancy is a disease paradigm with a nearly immeasurable scope. However, another disease paradigm has a similarly large sphere – infectious diseases. This project's goal is to determine whether immunotherapy findings in a cancer context can span paradigms and similarly impact treatment approaches in an infectious disease context. Our goal is to perform infectious disease-related experiments without the need to incorporate fully infectious agents into our experimental approach. To do this, we obtained cells that express human immunodeficiency virus (HIV) envelope proteins constitutively. HIV was chosen as the pathogen to represent the infectious disease paradigm in our studies because HIV treatments exist but are not curative (yet). Our goal is to contribute to many research efforts focused on helping to “train” the immune system to fight HIV in the absence of other treatments (e.g., antiretroviral therapy). The target cells with HIV protein on their surface appear to be infected from the perspective of the human NK cells that we have treated with immunotherapies. To allow the NK cells to recognize the presence of HIV protein, we use an antibody that is specific for the envelope and capable of directing the NK cell to perform the killing function known as antibody-dependent cell-mediated cytotoxicity (ADCC). Data to date will be presented.