Glycosaminoglycan Accumulation Increases the Susceptibility of Aortic Media to Dissection

Presenter Information

Ramin ShahbadFollow

Presenter Type

UNO Graduate Student (Doctoral)

Major/Field of Study

Biomechanics

Other

Biomechanics

Advisor Information

Anastasia Desyatova

Location

CEC RM #128

Presentation Type

Oral Presentation

Start Date

22-3-2024 10:30 AM

End Date

22-3-2024 11:45 AM

Abstract

Aortic dissection is a critical medical condition characterized by the separation of arterial inner walls, predominantly affecting the thoracic aorta. This phenomenon initiates with an initial intimal tear followed by the propagation of a crack through the medial layer, either longitudinally or circumferentially along the aorta. Despite extensive research, the precise relationship between arterial wall architecture and aortic dissection remains elusive. Glycosaminoglycans (GAGs), prominent histological features of arterial walls, are hypothesized to increase Donnan swelling pressure between elastic lamellae, potentially contributing to aortic dissection. Therefore, this study investigates the influence of GAG accumulation in the aortic media on the force required for aortic dissection. We conducted a series of 61 peeling tests on human thoracic aorta strips obtained from 35 tissue donors aged 13 to 76 years (average age 38±15, with 34% female representation). Additionally, GAG density was quantified using Movat's Pentachrome stains on the peeled sections. Our results demonstrate a significant decrease in delamination resistance (force per strip width) with age (p

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Mar 22nd, 10:30 AM Mar 22nd, 11:45 AM

Glycosaminoglycan Accumulation Increases the Susceptibility of Aortic Media to Dissection

CEC RM #128

Aortic dissection is a critical medical condition characterized by the separation of arterial inner walls, predominantly affecting the thoracic aorta. This phenomenon initiates with an initial intimal tear followed by the propagation of a crack through the medial layer, either longitudinally or circumferentially along the aorta. Despite extensive research, the precise relationship between arterial wall architecture and aortic dissection remains elusive. Glycosaminoglycans (GAGs), prominent histological features of arterial walls, are hypothesized to increase Donnan swelling pressure between elastic lamellae, potentially contributing to aortic dissection. Therefore, this study investigates the influence of GAG accumulation in the aortic media on the force required for aortic dissection. We conducted a series of 61 peeling tests on human thoracic aorta strips obtained from 35 tissue donors aged 13 to 76 years (average age 38±15, with 34% female representation). Additionally, GAG density was quantified using Movat's Pentachrome stains on the peeled sections. Our results demonstrate a significant decrease in delamination resistance (force per strip width) with age (p